Tubulointerstitial nephritis ICD code 10. Chronic interstitial nephritis - description, causes, symptoms (signs), diagnosis, treatment

Pyelonephritis is a nonspecific inflammatory disease of an infectious nature, which affects the pyelocaliceal system and interstitial tissue. In 20% of cases, this pathology develops secondary to acute inflammation. Most often the lesion is bilateral. The risk group includes young girls and women, which is associated with easier penetration of microbes from the urethra and bladder. For chronic pyelonephritis, the ICD-10 code is N11.

Pyelonephritis

Types of diagnoses

All urologists know about pyelonephritis. The following types of this pathology are distinguished in children and adults:

1. Chronic obstructive (code N11.1).
2. Non-obstructive, caused by reflux (reflux of urine from the ureters). The ICD-10 code is N11.0.
3. Unspecified etiology (code N11.9).
4. Infectious.
5. Non-infectious.

If a person is diagnosed with pyelonephritis, the ICD-10 code will depend on the etiology of the disease and the results of instrumental and laboratory tests.

Features of chronic pyelonephritis

This disease most often has a microbial (bacterial) nature. Chronic inflammation of the kidneys is caused by cocci, Escherichia coli, Proteus, Pseudomonas aeruginosa and other bacteria. This pathology is preceded by acute pyelonephritis. Predisposing factors for the development of chronic pyelonephritis (according to ICD-10 code N11) are:

• untimely and incorrect treatment of acute inflammation;
• foci of bacterial infection (tonsillitis, inflammation of the prostate, otitis media, inflammation of the paranasal sinuses, urethritis, cholecystitis);
• difficulty in the outflow of urine;
• stones;
• irrational (monotonous) nutrition;
• narrowing of the ureters;
• reflux;
• tumors;
• benign prostatic hyperplasia;
• diabetes;
• immunodeficiency states;
• intoxication of the body;
• childbirth and onset of sexual activity;
• congenital features of the development of the urinary organs (diverticula, spermatocele).

Chronic pyelonephritis

The disease is not as pronounced as acute pyelonephritis. Exacerbations, which occur mainly in the cold season, are replaced by remission. The following symptoms are characteristic of chronic pyelonephritis:

1. Low-grade fever.
2. Heaviness in the lower back.
3. It's a dull pain.
4. Disruption of the urination process (pain, frequent micturition).
5. Headache.
6. Fatigue during work.
7. Malaise.
8. Signs of arterial hypertension. Characteristic of the hypertensive form of pyelonephritis. Patients experience sudden increases in blood pressure, attacks of hypertensive crisis, severe headache, shortness of breath, nausea and dizziness. Sometimes there is pain in the heart area.
9. Positive symptom of lumbar concussion (Pasternatsky).
10. Signs of anemia.
11. Sleep disturbance.
12. Edema. Appears in advanced cases. They occur mainly in the first half of the day. The swelling is soft, symmetrical, mobile, pale, warm to the touch, localized on the face and lower extremities. They appear quickly and disappear just as quickly.

Objective signs of the disease are the presence of protein in the urine (proteinuria), excess of normal levels of leukocytes, the presence of columnar epithelium and bacteria. Sometimes blood appears in the urine. Often the disease is detected already at the stage of chronic renal failure.

Stages of tubulointerstitial pathology

Tubulointerstitial nephritis in ICD-10 is prescribed without stages. There are only 3 of them. Stage 1 is characterized by the following violations:

• infiltration of tissues with leukocytes;
• atrophic changes in the collecting ducts;
• intactness of the renal glomeruli.

At stage 2 of the disease, sclerotic changes are observed. Part of the interstitial tissue is replaced by scar tissue. Hyalinization of the glomeruli and vascular damage also occur. At stage 3, the kidney decreases in volume and shrinks. Its surface becomes lumpy. At this stage, symptoms of renal failure are severe.

Chronic pyelonephritis during pregnancy

The classification separately identifies the gestational form of the disease. Chronic pyelonephritis in pregnant women is much more common than in the rest of the population. This is due to hormonal changes and decreased immunity. In pregnant women, the tone of the urethra, ureters and bladder decreases, which facilitates the penetration of infection. An important factor is that many medications are contraindicated during pregnancy, which complicates the treatment of acute pyelonephritis and contributes to the transition of the disease to a chronic form.

The development of the disease is facilitated by increased pressure on the urinary organs by an enlarged uterus and impaired urine outflow. Pyelonephritis (ICD-10 code N11) in pregnant women is often asymptomatic. Complaints are observed only during exacerbations. Changes are detected during a general urine test.

Chronic kidney inflammation during pregnancy can lead to the following consequences:

• arterial hypertension;
• renal failure;
• gestosis (toxicosis).

Chronic pyelonephritis during pregnancy

It still seems to you that it is impossible to regain potency

Chronic and acute pyelocystitis, pyelitis and cystopyelonephritis can negatively affect potency. To avoid this, you need to treat the disease in a timely manner. Complex therapy includes:

1. Following a strict diet with limited salt. Patients are advised to eat fermented milk products, vegetables, fruits, berries (watermelons), drink juices, fruit drinks and herbal infusions. Alcoholic drinks, coffee, pickles, smoked meats, spices, fatty and spicy foods are excluded from the menu.
2. Taking antibacterial agents. They are indicated in the acute phase. For pyelonephritis, fluoroquinolones (Nolitsin), penicillins (Amoxiclav), cephalosporins (Suprax, Ceftriaxone), aminoglycosides and nitrofurans (Furadonin) are used.
3. The use of symptomatic drugs (antihypertensives, antispasmodics).
4. Physiotherapy (SMT therapy, ultrasound, chloride baths).

The nature and severity of clinical manifestations of AIN depend on the severity of general intoxication of the body and on the degree of activity of the pathological process in the kidneys. The first subjective symptoms of the disease usually appear 2-3 days after the start of treatment with antibiotics (most often penicillin or its semi-synthetic analogues) due to exacerbation of chronic tonsillitis, tonsillitis, otitis, sinusitis, ARVI and other diseases that precede the development of AIN. In other cases, they occur several days after the prescription of nonsteroidal anti-inflammatory drugs, diuretics, cytostatics, administration of radiocontrast agents, serums, and vaccines. Most patients complain of general weakness, sweating, headache, aching pain in the lumbar region, drowsiness, decreased or loss of appetite, and nausea. Often the mentioned symptoms are accompanied by chills with fever, muscle aches, sometimes polyarthralgia, and allergic skin rashes. In some cases, moderate and short-term arterial hypertension may develop. Edema is not typical for AIN and is usually absent. Dysuric phenomena are not usually observed. In the vast majority of cases, polyuria with low relative density of urine (hyposthenuria) is noted from the first days. Only in very severe AIN at the onset of the disease is there a significant decrease (oliguria) in urine up to the development of anuria (combined, however, with hyposthenuria) and other signs of ARF. At the same time, urinary syndrome is also detected: slight (0.033-0.33 g/l) or (less often) moderately expressed (from 1.0 to 3.0 g/l) proteinuria, microhematuria, slight or moderate leukocyturia, cylindruria with a predominance of hyaline, and in severe cases - the appearance of granular and waxy cylinders. Oxalaturia and calciuria are often detected.
  The origin of proteinuria is associated primarily with a decrease in protein reabsorption by the epithelium of the proximal tubules, but the possibility of secretion of a special (specific) tissue protein Tamm-Horsfall into the lumen of the tubules is not excluded (B. I. Shulutko, 1983).
  The mechanism of microhematuria is not entirely clear.
  Pathological changes in urine persist throughout the disease (for 2-4-8 weeks). Polyuria and hyposthenuria last especially long (up to 2-3 months or more). Oliguria, which is sometimes observed in the first days of the disease, is associated with an increase in intratubular and intracapsular pressure, which leads to a drop in effective filtration pressure and a transient decrease in glomerular filtration rate. Along with a decrease in concentration ability, a violation of the nitrogen excretory function of the kidneys develops early (also in the first days) (especially in severe cases), which is manifested by hyperazotemia, i.e., an increase in the level of urea and creatinine in the blood. It is typical that hyperazotemia develops against the background of polyuria and hyposthenuria. It is also possible to have a disorder of electrolyte balance (hypokalemia, hyponatremia, hypochloremia) and acid-base balance with symptoms of acidosis. The severity of the mentioned kidney disorders in the regulation of nitrogen balance, acid-base balance and water-electrolyte homeostasis depends on the severity of the pathological process in the kidneys and reaches its greatest extent in the case of acute renal failure.
  As a consequence of the inflammatory process in the kidneys and general intoxication, characteristic changes in the peripheral blood are observed: slight or moderate leukocytosis with a slight shift to the left, often eosinophilia, an increase in ESR. In severe cases, anemia may develop. A biochemical blood test reveals C-reactive protein, elevated levels of DPA test, sialic acids, fibrinogen (or fibrin), dysproteinemia with hyper-a1- and a2-globulinemia.
  When assessing the clinical picture of AIN and its diagnosis, it is important to keep in mind that in almost all cases, and already in the first days from the onset of the disease, signs of renal failure of varying severity develop: from a slight increase in the blood level of urea and creatinine (in mild cases) to typical picture of acute renal failure (in severe cases). It is characteristic that the development of anuria (severe oliguria) is possible, but not at all necessary. More often, renal failure develops against the background of polyuria and hyposthenuria.
  In the vast majority of cases, the symptoms of renal failure are reversible and disappear after 2-3 weeks, but the impairment of the concentration function of the kidneys persists, as already noted, for 2-3 months or more (sometimes up to a year).
  Taking into account the characteristics of the clinical picture of the disease and its course, the following variants (forms) of IIN are distinguished (B.I. Shulutko, 1981).
  1. An expanded form, which is characterized by all the above clinical symptoms and laboratory signs of this disease.
  2. A variant of AIN, which occurs according to the type of “banal” (ordinary) acute renal failure with prolonged anuria and increasing hyperazotemia, with the phasic development of the pathological process characteristic of acute renal failure and its very severe course, requiring the use of acute hemodialysis when providing care to the patient.
  3. “Abortive” form with its characteristic absence of anuria phase, early development of polyuria, slight and short-term hyperazotemia, favorable course and rapid restoration of nitrogen excretory and concentration (within 1-1.5 months) kidney functions.
  4. “Focal” form, in which the clinical symptoms of AIN are weakly expressed, erased, changes in urine are minimal and inconsistent, hyperazotemia is either absent or insignificant and quickly transient. This form is more characterized by acutely occurring polyuria with hyposthenuria, rapid (within a month) restoration of the concentration function of the kidneys and the disappearance of pathological changes in the urine. This is the easiest option and the most favorable outcome for IUI. In outpatient settings, it usually passes as an “infectious-toxic kidney.”
  With AIN, the prognosis is most often favorable. Typically, the disappearance of the main clinical and laboratory symptoms of the disease occurs in the first 2-4 weeks from its onset. During this period, urine and peripheral blood parameters normalize, normal levels of urea and creatinine in the blood are restored, and polyuria with hyposthenuria persists much longer (sometimes up to 2-3 months or more). Only in rare cases, with a very severe course of AIN with pronounced symptoms of acute renal failure, an unfavorable outcome is possible. Sometimes AIN can become chronic, mainly due to its late diagnosis and improper treatment, and patients’ non-compliance with medical recommendations.

More than half a century has passed since the first successful kidney transplant was performed. Today, in transplantology, this operation is performed more often than others. More than ten thousand such operations are performed annually in the United States, and about 1000 in Russia, extending the lives of many people, including infants, by 6-20 years. Over more than 50 years of practice, a clear methodology has been developed, so a kidney transplant takes place step by step and is clearly timed.

General information

Kidney transplantation is a surgical operation to transplant an organ from a donor (living or a cadaveric kidney) to a patient. Sometimes, while leaving its own organ, the new kidney is transplanted to the same place, nearby, but most often it is placed in the iliac region. When transplanting from an adult to a child whose weight does not exceed 20 kg, the kidney is placed in the child’s abdominal cavity.

Pay attention! As a rule, the patient’s diseased native organ is left; it is removed only in some cases (for example, if its size is too large, polycystic disease), when there is not enough space to place a donor kidney nearby.

A kidney transplant operation requires mandatory preliminary preparation of the patient and the donor organ. After the kidney is removed from the donor, it is prepared, frozen, and placed in a special container. Immediately before the operation, they are washed and then placed in the patient’s body, quickly placing vessels, nerves and ureters (which may also be donor).

For reference: According to the International Classification of Diseases (ICD), there is a special coding for each disease, and kidney transplantation also has its own codes according to ICD-10. According to this coding, code Z52.4 indicates the kidney donor, code Z94.0 indicates the presence of the transplanted kidney, and code T86.1 indicates graft rejection or complications after surgery.

Indications for transplantation

A kidney transplant is indicated only when it is impossible to restore the functions of this organ, that is, in the thermal stage of chronic renal failure. This condition can occur as a result of many diseases, including:

• Trauma to the urinary organ;
• Congenital defects, anomalies;
• Chronic form of pyelonephritis or glomerulonephritis;
• Renal polycystic disease;
• Diabetic nephropathy;
• Nephritis, due to the development of lupus erythematosus and other diseases.

Renal replacement therapy, in the form of peritoneal dialysis and hemodialysis, can be administered to the patient for several years. This therapy involves kidney transplantation. Thanks to an organ transplant, provided that survival rate is good, the patient can live a full life for several years, without the need to undergo hemodialysis every few days. Kidney transplantation for children is especially urgent due to the fact that the procedure of blood purification by hemodialysis seriously slows down the development of the child.

Contraindications for transplantation

Today, there are a number of absolute contraindications for transplantation and a number of relative ones. Relative diseases include diseases that can potentially provoke complications after surgery, including:

• hemolytic uremic syndrome;
• membranous-proliferative glomerulonephritis;
• metabolic disorders that provoke deposits in the renal structure (for example, gout), etc.

Kidney transplantation is not performed if the following absolute contraindications exist:

• Recent removal of a cancerous tumor or its presence;
• Severe active infections (eg, HIV or tuberculosis);
• Chronic diseases in their acute or severe form;
• Immunological cross-reaction with donor lymphocytes in this patient;
• Decompensated stage of cardiovascular diseases;
• Serious personality changes due to which the patient will not be able to adapt after organ transplantation.

Pay attention! Diabetes mellitus and inactive forms of hepatitis B and C are not contraindications for surgery. Simultaneously with the kidneys, during one operation, a pancreas can be transplanted (which is important for patients with diabetes).

Types and compatibility of transplants

Kidney transplantation occurs with an organ obtained from a corpse or from a living person (usually a relative). In the second case, the survival rate is quite high with complete restoration of functions. Compatibility is determined by three main parameters:

• compatibility of alleles of the HLA genes of the donor and the patient who will undergo the transplant;
• matching the blood group of the recipient and the donor;
• matching by age, weight, gender. Preferred, but not always followed.

According to statistics, the survival rate of a recipient with an organ taken from a living person is 98%, the survival rate of the organ itself is 94% of cases. With a kidney removed from a corpse, patients survive in 94% of cases, and the transplant itself takes root in 88% of cases.

Pay attention! The safest transplant is considered to be a related “living” transplant, where the donor is a living relative. However, not all relatives who are able to donate a kidney without harm to their health have the same blood type and level of leukocyte agents (HLA study).

The donor must not have the following diseases:

• hepatitis B and C in acute form;
• HIV and AIDS;
• tuberculosis;
• venereal diseases;
• helminthic infestations.

Taking into account all these requirements, the circle of potential donors is significantly narrowed. Transplantologists propose expanding the criteria by posthumously removing kidneys and using organs from older people who died from pathologies of other organs. However, these methods are met with disapproval among people.

Cadaveric kidneys are removed immediately after the biological death of the donor. Such a transplant, according to one of the methods, is cleared of blood and connected to a special apparatus for artificial pumping of fluids, then continuously washed with a preservative solution (Viaspan, EuroCollins, UW, Custodiol). Another, less expensive method uses a triple bag system for storage no higher than 5-6°C. To do this:

1. The organ, cleared of blood, is placed in a sterile bag with a preservative solution;
2. This package is placed in the second, with a layer of sterile snow;
3. The outer third bag is filled with ice-cold saline.

The best graft survival rates are observed when transplanted in the first 24 hours after removal, but the organ can remain in these conditions for up to 72 hours. As a rule, surgery is performed as soon as a suitable organ becomes available. The recipient can remain at home or in the hospital all this time, waiting for his turn. If a kidney was received from a living donor, it survives much better than a cadaveric one. This is because the organ did not suffer from cold ischemia and the donor was thoroughly examined.

Today in the Russian Federation, kidney transplantation is permitted only from a capable close relative who has given his voluntary consent to the removal and transplantation of an organ who is between the ages of 18 and 65 years.

Preparation and necessary tests

Kidney transplantation requires special preparation. A group of specialists prepares the recipient for the operation: a surgeon, an anesthesiologist, a nephrologist-transplantologist, junior medical staff, a psychologist, and even a nutritionist. If the donor is a living person, then the preparation can be thorough and lengthy, and in the case of a cadaveric kidney, the patient may be called to the clinic urgently (according to the queue on the transplant waiting list). A number of special compatibility tests are carried out (especially in the case of a cadaveric organ), and if there is a high risk of rejection, the patient may be asked to wait for the next more suitable organ.

Mandatory laboratory and instrumental tests performed before surgery include:

• Blood test: for creatine, urea level, hemoglobin, calcium level, potassium level, etc.;
• X-ray or ultrasound;
• Hemodialysis (carried out in the absence of contraindications in adults; usually not performed in children).

Period after surgery

To suppress the immune reaction to the transplant, special drugs are prescribed on the day of surgery (for example, prednisolone, mifortic, cyclosporine), which significantly increases the survival rate of the organ. Taking these immunosuppressants can continue for up to 3-6 months after transplantation.

The next day after the operation, the patient is allowed to walk. The period of hospital stay is about 1-2 weeks, after which the patient with a new kidney is sent home, with mandatory regular home measurements of body temperature, blood pressure, etc. In addition, it is necessary to monitor body weight, follow a special diet, and control diuresis.

During the first visit to the attending physician after discharge, the sutures are removed (approximately 10-14 days after discharge). A clinical examination is carried out every 2 weeks, then less often and until the end of life it is necessary to visit the attending physician at least once a month.

During the clinical examination the following is carried out:

• Checking blood pressure levels;
• Checking the density of the transplanted organ;
• The murmur of blood vessels over the graft can be heard;
• Diuresis is checked;
• General urine analysis and daily protein;
• Blood test for biochemistry and general;
• Twice a year it is necessary to donate blood for uric acid and lipids;
• At least once a year, an ECG, ultrasound, fluorography and other necessary types of research are performed.

Life after transplant

Patients with chronic renal failure get up to 20 years of full life with a new organ. In the case of a cadaveric kidney, a person receives an additional 6 to 10 years of life, and in the case of an organ from a living person (relative) - 15-20 years.

After transplantation, a special diet with a reduced salt and sugar content is recommended, the consumption of baked goods is reduced, and smoked and fried foods should be avoided. The volume of liquid per day is also limited to 1.5-2 liters. Dietary table No. 7 is considered optimal.

After the transplant, you should not lift weights (up to 5 kg, and after 6 months - up to 10 kg) and intense physical activity. However, moderate physical exercise and stress are encouraged and considered beneficial during the rehabilitation period (especially during cadaveric kidney transplantation).

Important. In addition, it is necessary to exclude sexually transmitted infections that require serious treatment. For these purposes, barrier contraception is recommended. It is possible to become pregnant after a transplant, but only after consulting with your doctor and obstetrician-gynecologist to assess all possible risks.

Possible complications

The most important complication after a transplant is organ rejection. Experts distinguish three types of rejection:

1. Super sharp. Occurs 1 hour after the end of the operation. An extremely rare case;
2. Spicy. Occurs in the postoperative period, 5-21 days after surgery;
3. Chronic. There are no time limits.

Basically, signs that the kidney is not taking root appear gradually and this process can be stopped with the help of medications. However, if, when the kidney fails to function, the chronic rejection syndrome continues to increase, then retransplantation is required, that is, a new transplant.

Other possible complications include:

• Urological nature (hypertension, thrombosis, bleeding, stenosis of arteries in the transplanted kidney, etc.);
• Vascular in nature (hematuria, blockage of the lumen of the ureter, etc.)

Just like after any other operation, infection of the postoperative suture is also considered a possible complication.

Features of microhematuria

If the number of red blood cells in the urine becomes too high, this indicates the appearance of hematuria. There are 2 types of this disease: microhematuria and macrohematuria.

In the second case, the urine changes color, resulting in the shade of meat slop. And the first variant of the disease is determined only with the help of a laboratory. It cannot be determined visually; no symptoms are observed.

The difference between types of pathology

There are two types of hematuria: microscopic and macroscopic. Doctors can determine the macroscopic form using the changed color of urine, and microhematuria only based on test results. Thus, the difference between these methods is in defining the disease. But in the first and second variants, the appearance of blood in the urine is a violation of the genitourinary system.

Reasons for appearance

To find out about the onset of the disease, you need to take a urine test. A micro-examination of urine sediment is carried out. As a result, the patient becomes aware of elevated red blood cells. This indicates the initial stage of hematuria or a minor form.

In such cases, it is necessary to undergo a full examination, determine and treat the cause of the disease. In most cases, in parallel with this disease, mild renal and urethral disorders appear.

A consequence of the appearance of hematuria is also diffuse or focal nephritis, as well as various infections. The development of the disease often occurs due to an enlarged prostate or after taking blood thinning medications.

Cystoscopy is performed when the initial stage of a tumor process has formed in the bladder. Microhematuria is more common among employees of paint and varnish and aniline companies. Its known signs are the following manifestations:

• swelling of the face;
• discomfort during urination.

Symptoms of the disease

As the disease develops, a person goes to the toilet very often, and experiences pain. This indicates inflammation of the urinary system.

If pain occurs in the abdomen or sides, this indicates the appearance of kidney nephropathy or ureteral disease. In such cases, the temperature usually rises, which indicates the formation of inflammation or injury to the kidneys, as well as the development of a malignant tumor.

If hematuria has the listed symptoms, then it is necessary to undergo a bacteriological examination of urine to determine the cause of their occurrence. If urination is impaired during the development of the disease, bladder carcinomas appear. To identify them, a cytological examination is required.

Microhematuria in men

This disease is considered quite common among men. But it is not the main disease, but a symptom of a rather serious illness. The person himself rarely determines this symptom; only qualified doctors can do this using tests to determine the number of red blood cells. The reasons why the disease occurs in men are as follows:

• benign prostate tumor;
• urolithiasis;
• prostate oncology;
• bladder injury;
• anemia;
• kidney nephropathy;
• varicose veins;
• urethral polyps;

• birth defects;
• the presence of blood clots in the body;
• poor blood clotting;
• urinary tract infection;
• viral infections;
• high blood pressure;
• physical overload;
• intoxication in the body.

Regardless of the reason, a man must come to an appointment with a urologist. If you ignore the disease, the consequences can be more than serious. As prescribed by the doctor, the patient undergoes all the necessary tests, after which conservative treatment is individually selected.

If the disease is mild, then therapy is carried out with the help of medications. But if the disease threatens a man’s life, then surgery is necessary.

Treatment of the disease in men

The goal of treatment is to eliminate the causes of the disease. Features of therapy are:

• To eliminate inflammation, it is necessary to take antibacterial drugs. The duration of therapy, type of drug and dosage are also prescribed by the doctor;
• to identify the urethra or stone in the urinary tract, antispasmodic drugs and thermal procedures are prescribed;
• to stop bleeding, you need to take medications such as Dicynone, aminocaproic acid and Vikasol;
• if protein increases with hematuria, the doctor prescribes corticosteroids;
• in case of infection, take antibiotics and undergo regular tests;
• if there is kidney nephropathy, surgical intervention is necessary;
• if microhematuria is chronic, you need to take B vitamins and medications that help increase iron;
• strict adherence to bed rest.

The treatment method for each person will be prescribed individually.

In addition to medications, you should also turn to traditional medicine. Decoctions of plants such as nettle and yarrow are more effective. They are not inferior to bearberry leaves and barley seeds.

The examination must be carried out immediately, and then therapy must be carried out, otherwise the disease will develop and in some cases develop into cancer.

ICD code

ICD is an international classification of diseases. It was created by the World Health Organization to code diagnoses. It is intended for standard healthcare assessment.

The disease has the following codes in this classification:

• N02–9 - basic hematuria with characteristic modifications;
• R31 - nonspecific hematuria.

Disease in children

It is easier to identify this disease in a child than in an adult. This is because his parents take him for a full examination every year. If microhematuria is detected in children, this indicates the appearance of diseases of the genitourinary system, injuries to internal organs or kidney disease.

The known causes of this disease in childhood are:

• acute cystitis;
• congenital defects of the bladder and kidneys;
• bladder injury;
• disturbance in blood circulation;
• side effect from taking medications;
• vascular pathology;
• urolithiasis;
• papilloma of the bladder;
• foreign body in the urethra.

A nephrologist prescribes therapy for the child. The method of treatment will depend on the test results and the causes of the disease. Before prescribing treatment, the doctor is obliged to check the intolerance of a particular drug, as well as the child’s health status. How long the therapy will last is determined by the doctor.

Often the doctor prescribes a course of antibiotics and bed rest. If the form is more severe, then surgery cannot be avoided.

Once the diagnosis is made, the doctor is obliged to identify the cause of its occurrence. This is required for proper treatment and elimination of this process.

Treatment of the disease in children

If the analysis reveals Pseudomonas aeruginosa in the child’s urine, the doctor is obliged to prescribe treatment with antibiotics.

An effective drug is Ceftriaxone. During treatment with this medicine, the child will go to the toilet less often. You cannot give medications yourself, only as prescribed by a doctor, otherwise increased sensitivity of the organs often occurs.

You must follow a strict diet. Large amounts of fried, salted, smoked food, as well as chemical food additives and vitamins are prohibited.

After the course of treatment, all urine and blood tests are required again. Regular visits to a nephrologist are also necessary.

As for treatment with herbs, decoctions of yarrow and nettle are effective, as well as rosehip, blackberry and peony root and juniper. Before taking these decoctions, you should consult your doctor.

Renal hematuria occurs when the kidneys and venous outflow are impaired.

Pregnancy

The formation of microhematuria during pregnancy in women is observed at 2–3 months. Intense growth of the fetus has a bad effect on the functioning of the kidneys, and also compresses the ureter of the uterus.

If urine stagnates in the pelvis, stones can form, which subsequently damage the epithelium and contribute to bleeding in women.

It is important not to confuse bleeding from the urinary tract with bleeding from the uterus. If blood appears from the uterus, then this is a threat to the child and the mother.

Microhematuria often occurs due to the use of anticoagulants. The risk of developing this disease also increases if women had kidney inflammation before pregnancy or with pyelonephritis, which is chronic.

CLASS XIV. DISEASES OF THE GINOROGENITAL SYSTEM (N00-N99)

This class contains the following blocks:
N00-N08 Glomerular diseases
N10-N16 Tubulointerstitial kidney disease
N17-N19 Kidney failure
N20-N23 Urolithiasis
N25-N29 Other diseases of the kidney and ureter
N30-N39 Other diseases of the urinary system
N40-N51 Diseases of the male genital organs
N60-N64 Breast diseases
N70-N77 Inflammatory diseases of the female pelvic organs
N80-N98 Non-inflammatory diseases of the female genital organs
N99 Other disorders of the genitourinary system

The following categories are marked with an asterisk:
N08*Glomerular lesions in diseases classified elsewhere
N16* Tubulointerstitial kidney damage in diseases classified elsewhere
N22* Urinary tract stones in diseases classified elsewhere
N29* Other lesions of the kidney and ureter in diseases classified elsewhere
N33* Lesions of the bladder in diseases classified elsewhere
N37* Lesions of the ureter in diseases classified elsewhere
N51* Lesions of the male genital organs in diseases classified in other headings
N74* Inflammatory lesions of the pelvic organs in women with diseases classified in other headings
N77* Ulceration and inflammation of the vulva and vagina in diseases classified elsewhere

GLOMEROULAR DISEASES (N00-N08)

If necessary, identify an external cause (Class XX) or if renal failure is present ( N17-N19) use additional code.

Excluded: hypertension with predominant kidney damage ( I12. -)

With rubrics N00-N07 the following fourth digits may be used to classify morphological changes. Categories 0-.8 should not be used unless specific studies have been performed to identify the lesions (eg, renal biopsy or autopsy). Three-digit rubrics are based on clinical syndromes.

0 Minor glomerular abnormalities. Minimal damage
.1 Focal and segmental glomerular lesions
Focal and segmental:
hyalinosis
sclerosis
Focal glomerulonephritis
.2 Diffuse membranous glomerulonephritis
.3 Diffuse mesangial proliferative glomerulonephritis
.4 Diffuse endocapillary proliferative glomerulonephritis
.5 Diffuse mesangiocapillary glomerulonephritis. Membranous proliferative glomerulonephritis (types 1 and 3 or NOS)
.6 Dense sediment disease. Membranous proliferative glomerulonephritis (type 2)
.7 Diffuse crescentic glomerulonephritis. Extracapillary glomerulonephritis
.8 Other changes. Proliferative glomerulonephritis NOS
.9 Unspecified change

N00 Acute nephritic syndrome

Included: acute:
glomerular disease
glomerulonephritis
nephritis
renal disease NOS
Excluded: acute tubulointerstitial nephritis ( N10)
nephritic syndrome NOS ( N05. -)

N01 Rapidly progressive nephritic syndrome

Included: rapidly progressive:
glomerular disease
glomerulonephritis
nephritis
Excludes: nephritic syndrome NOS ( N05. -)

N02 Recurrent and persistent hematuria

Included: hematuria:
benign (family) (children's)
with morphological lesion, specified in 0-.8
Excludes: hematuria NOS ( R31)

N03 Chronic nephritic syndrome

Included: chronic(s):
glomerular disease
glomerulonephritis
nephritis
renal disease NOS
Excluded: chronic tubulointerstitial nephritis ( N11. -)
N18. -)
nephritic syndrome NOS ( N05. -)

N04 Nephrotic syndrome

Includes: congenital nephrotic syndrome
lipoid nephrosis

N05 Nephritic syndrome, unspecified

Includes: glomerular disease)
glomerulonephritis) NOS
jade)
nephropathy NOS and renal disease NOS with morphological lesion specified in clause 0-.8
Excludes: nephropathy NOS of unknown cause ( N28.9)
renal disease NOS of unknown cause ( N28.9)
tubulointerstitial nephritis NOS ( N12)

N06 Isolated proteinuria with specified morphological lesion

Includes: proteinuria (isolated) (orthostatic)
(persistent) with morphological lesion, specified
v.0-.8
Excluded: proteinuria:
NOS ( R80)
Bence-Jones ( R80)
caused by pregnancy ( O12.1)
isolated NOS ( R80)
orthostatic NOS ( N39.2)
persistent NOS ( N39.1)

N07 Hereditary nephropathy, not elsewhere classified

Excludes: Alport syndrome ( Q87.8)
hereditary amyloid nephropathy ( E85.0)
syndrome (absence) (underdevelopment) of nails-patella ( Q87.2)
hereditary familial amyloidosis without neuropathy ( E85.0)

N08* Glomerular lesions in diseases classified elsewhere

Includes: nephropathy in diseases classified elsewhere
Excluded: renal tubulointerstitial lesions in diseases classified elsewhere ( N16. -*)

Included: pyelonephritis
Excluded: cystic pyeloureteritis ( N28.8)

N10 Acute tubulointerstitial nephritis

Spicy:

pyelitis
pyelonephritis
B95-B97).

N11 Chronic tubulointerstitial nephritis

Included: chronic:
infectious interstitial nephritis
pyelitis
pyelonephritis
B95-B97).

N11.0 Non-obstructive chronic pyelonephritis associated with reflux
Pyelonephritis (chronic) associated with (vesicoureteral) reflux
Excludes: vesicoureteral reflux NOS ( N13.7)
N11.1 Chronic obstructive pyelonephritis
Pyelonephritis (chronic) associated with:
anomaly) (ureteropelvic
bend) (connections
obstruction) (pelvic segment of the ureter
structure) (ureter
Excluded: calculous pyelonephritis ( N20.9)
obstructive uropathy ( N13. -)
N11.8 Other chronic tubulointerstitial nephritis
Non-obstructive chronic pyelonephritis NOS
N11.9 Chronic tubulointerstitial nephritis, unspecified
Chronic:
interstitial nephritis NOS
pyelitis NOS
pyelonephritis NOS

N12 Tubulointerstitial nephritis, not specified as acute or chronic

Interstitial nephritis NOS
Pyelitis NOS
Pyelonephritis NOS
Excluded: calculous pyelonephritis ( N20.9)

N13 Obstructive uropathy and reflux uropathy

Excluded: kidney and ureteral stones without hydronephrosis ( N20. -)
congenital obstructive changes in the renal pelvis and ureter ( Q62.0-Q62.3)
obstructive pyelonephritis ( N11.1)

N13.0 Hydronephrosis with obstruction of the ureteropelvic junction
Excluded: with infection ( N13.6)
N13.1 Hydronephrosis with ureteral stricture, not elsewhere classified
Excluded: with infection ( N13.6)
N13.2 Hydronephrosis with obstruction of the kidney and ureter by a stone
Excluded: with infection ( N13.6)
N13.3 Other and unspecified hydronephrosis
Excluded: with infection ( N13.6)
N13.4 Hydroureter
Excluded: with infection ( N13.6)
N13.5 Kink and stricture of the ureter without hydronephrosis
Excluded: with infection ( N13.6)
N13.6 Pyonephrosis
Conditions listed in categories N13.0-N13.5, with infection. Obstructive uropathy with infection
If it is necessary to identify the infectious agent, use an additional code ( B95-B97).
N13.7 Uropathy due to vesicoureteral reflux
Vesicoureteral reflux:
NOS
with scarring
Excludes: pyelonephritis associated with vesicoureteral reflux ( N11.0)
N13.8 Other obstructive uropathy and reflux uropathy
N13.9 Obstructive uropathy and reflux uropathy, unspecified. Urinary tract obstruction NOS

N14 Tubulointerstitial and tubular lesions caused by drugs and heavy metals

If it is necessary to identify a toxic substance, use an additional external cause code (Class XX).

N14.0 Analgesic-induced nephropathy
N14.1 Nephropathy caused by other drugs, medications or biologically active substances
N14.2 Nephropathy caused by unspecified drug, medicament and biologically active substance
N14.3 Nephropathy caused by heavy metals
N14.4 Toxic nephropathy, not elsewhere classified

N15 Other tubulointerstitial kidney diseases

N15.0 Balkan nephropathy. Balkan endemic nephropathy
N15.1 Abscess of the kidney and perinephric tissue
N15.8 Other specified tubulointerstitial kidney lesions
N15.9 Tubulointerstitial kidney damage, unspecified. Kidney infection NOS
Excludes: urinary tract infection NOS ( N39.0)

N16* Tubulointerstitial lesions of the kidneys in diseases classified elsewhere

leukemia ( C91-C95+)
lymphoma ( C81-C85+, C96. -+)
multiple myeloma ( C90.0+)
N16.2* Tubulointerstitial kidney damage due to blood diseases and disorders involving the immune mechanism
Tubulointerstitial kidney damage with:
mixed cryoglobulinemia ( D89.1+)
sarcoidosis ( D86. -+)
N16.3* Tubulointerstitial kidney damage due to metabolic disorders
Tubulointerstitial kidney damage with:
cystinosis ( E72.0+)
glycogen storage diseases ( E74.0+)
Wilson's disease ( E83.0+)
N16.4* Tubulointerstitial kidney damage in systemic connective tissue diseases
Tubulointerstitial kidney damage with:
[Sjögren's] sicca syndrome ( M35.0+)
systemic lupus erythematosus ( M32.1+)
N16.5* Tubulointerstitial kidney damage due to transplant rejection ( T86. -+)
N16.8* Tubulointerstitial kidney damage in other diseases classified elsewhere

RENAL FAILURE (N17-N19)

If it is necessary to identify an external agent, use an additional external cause code (class XX).

Excluded: congenital renal failure ( P96.0)
tubulointerstitial and tubular lesions caused by drugs and heavy metals ( N14. -)
extrarenal uremia ( R39.2)
hemolytic-uremic syndrome ( D59.3)
hepatorenal syndrome ( K76.7)
postpartum ( O90.4)
prerenal uremia ( R39.2)
renal failure:
complicating abortion, ectopic or molar pregnancy ( O00-O07, O08.4)
after childbirth and delivery ( O90.4)
after medical procedures ( N99.0)

N17 Acute renal failure

N17.0 Acute renal failure with tubular necrosis
Tubular necrosis:
NOS
spicy
N17.1 Acute renal failure with acute cortical necrosis
Cortical necrosis:
NOS
spicy
renal
N17.2 Acute renal failure with medullary necrosis
Medullary (papillary) necrosis:
NOS
spicy
renal
N17.8 Other acute renal failure
N17.9 Acute renal failure, unspecified

N18 Chronic renal failure

Includes: chronic uremia, diffuse sclerosing glomerulonephritis
Excludes: chronic renal failure with hypertension ( I12.0)

N18.0 End stage kidney disease
N18.8 Other manifestations of chronic renal failure
Uremic neuropathy+ ( G63.8*)
Uremic pericarditis+ ( I32.8*)
N18.9 Chronic renal failure, unspecified

N19 Renal failure, unspecified

Uremia NOS
Excluded: renal failure with hypertension ( I12.0)
uremia of the newborn ( P96.0)

URILOSTICAL DISEASE (N20-N23)

N20 Kidney and ureteral stones

Excluded: with hydronephrosis ( N13.2)

N20.0 Kidney stones. Nephrolithiasis NOS. Concretions or stones in the kidney. Coral stones. Kidney stone
N20.1 Ureteral stones. Stone in the ureter
N20.2 Kidney stones with ureteral stones
N20.9 Urinary stones, unspecified. Calculous pyelonephritis

N21 Stones of the lower urinary tract

Included: with cystitis and urethritis

N21.0 Bladder stones. Stones in the diverticulum of the bladder. Bladder stone
Excluded: coral stones ( N20.0)
N21.1 Stones in the urethra
N21.8 Other stones in the lower urinary tract
N21.9 Stones in the lower urinary tract, unspecified

N22* Urinary tract stones in diseases classified elsewhere

N22.0* Urinary stones in schistosomiasis [bilharzia] ( B65. -+)
N22.8* Urinary tract stones in other diseases classified elsewhere

N23 Renal colic, unspecified

OTHER DISEASES OF THE KIDNEY AND URETER (N25-N29)

Excluded: with urolithiasis ( N20-N23)

N25 Disorders resulting from renal tubular dysfunction

Excludes: metabolic disorders classified under E70-E90

N25.0 Renal osteodystrophy. Azotemic osteodystrophy. Tubular disorders associated with phosphate loss
Renal:
rickets
dwarfism
N25.1 Nephrogenic diabetes insipidus
N25.8 Other disorders due to renal tubular dysfunction
Lightwood-Albright syndrome. Renal tubular acidosis NOS. Secondary hyperparathyroidism of renal origin
N25.9 Renal tubular dysfunction, specified

N26 Shriveled kidney, unspecified

Kidney atrophy (terminal). Renal sclerosis NOS
Excluded: wrinkled kidney with hypertension ( I12. -)
diffuse sclerosing glomerulonephritis ( N18. -)
hypertensive nephrosclerosis (arteriolar) (arteriosclerotic) ( I12. -)
small kidney for unknown reason ( N27. -)

N27 Small kidney of unknown origin

N27.0 Small kidney unilateral
N27.1 Small kidney bilateral
N27.9 Small kidney, unspecified

N28 Other diseases of the kidney and ureter, not elsewhere classified

Excludes: hydroureter ( N13.4)
kidney disease:
acute NOS ( N00.9)
chronic NOS ( N03.9)
kinking and stricture of the ureter:
with hydronephrosis ( N13.1)
without hydronephrosis ( N13.5)

N28.0 Renal ischemia or infarction
Renal artery:
embolism
obstruction
occlusion
thrombosis
Kidney infarction
Excluded: Goldblatt kidney ( I70.1)
renal artery (extrarenal part):
atherosclerosis ( I70.1)
congenital stenosis ( Q27.1)
N28.1 Acquired kidney cyst. Cyst (multiple) (single) kidney acquired
Excludes: cystic kidney disease (congenital) ( Q61. -)
N28.8 Other specified diseases of the kidneys and ureter. Kidney hypertrophy. Megaloureter. Nephroptosis
Pyelit)
Pyeloureteritis) cystic
Ureteritis)
Ureterocele
N28.9 Diseases of the kidney and ureter, unspecified. Nephropathy NOS. Kidney disease NOS
Excluded: nephropathy NOS and renal disorders NOS with morphological damage specified in sections 0-.8 ( N05. -)

N29* Other lesions of the kidney and ureter in diseases classified elsewhere

OTHER DISEASES OF THE URINARY SYSTEM (N30-N39)

Excluded: urinary tract infection (complicating):
O00 -O07 , O08.8 )
O23 . — , O75.3 , O86.2 )
with urolithiasis ( N20-N23)

N30 Cystitis

If necessary, identify the infectious agent ( B95-B97) or the corresponding external factor (class XX) use an additional code.
Excluded: prostatocystitis ( N41.3)

N30.0 Acute cystitis
Excluded: radiation cystitis ( N30.4)
trigonite ( N30.3)
N30.1 Interstitial cystitis (chronic)
N30.2 Other chronic cystitis
N30.3 Trigonite. Urethrotrigonitis
N30.4 Radiation cystitis
N30.8 Other cystitis. Bladder abscess
N30.9 Cystitis, unspecified

N31 Neuromuscular dysfunction of the bladder, not elsewhere classified

Excludes: spinal bladder NOS ( G95.8)
due to damage to the spinal cord ( G95.8)
neurogenic bladder associated with cauda equina syndrome ( G83.4)
urinary incontinence:
NOS ( R32)
updated ( N39.3-N39.4)

N31.0 Uninhibited bladder, not elsewhere classified
N31.1 Reflex bladder, not elsewhere classified
N31.2 Neurogenic bladder weakness, not elsewhere classified
Neurogenic bladder:
atonic (motor disturbances) (sensory disturbances)
autonomous
non-reflexive
N31.8 Other neuromuscular bladder dysfunctions
N31.9 Neuromuscular bladder dysfunction, unspecified

N32 Other lesions of the bladder

Excluded: bladder stone ( N21.0)
cystocele ( N81.1)
hernia or prolapse of the bladder in women ( N81.1)

N32.0 Obturation of the bladder neck. Bladder neck stenosis (acquired)
N32.1 Vesicointestinal fistula. Vesicocolic fistula
N32.2 Cystic fistula, not elsewhere classified
Excluded: fistula between the bladder and female genital tract ( N82.0-N82.1)
N32.3 Bladder diverticulum. Bladder diverticulitis
Excluded: stone in bladder diverticulum ( N21.0)
N32.4 Non-traumatic bladder rupture
N32.8 Other specified bladder lesions
Bladder:
calcified
wrinkled
N32.9 Bladder lesion, unspecified

N33* Lesions of the bladder in diseases classified elsewhere

N33.0* Tuberculous cystitis ( A18.1+)
N33.8* Lesions of the bladder in other diseases classified elsewhere
Lesions of the bladder due to schistosomiasis [bilharzia] ( B65. -+)

N34 Urethritis and urethral syndrome

If necessary, identify the infectious agent
use additional code ( B95-B97).
Excludes: Reiter's disease ( M02.3)
urethritis in diseases transmitted predominantly sexually ( A50-A64)
urethrotrigonitis ( N30.3)

N34.0 Urethral abscess
Abscess:
Cooper's glands
Littre glands
periurethral
urethral (glands)
Excluded: urethral caruncle ( N36.2)
N34.1 Nonspecific urethritis
Urethritis:
nongonococcal
non-venereal
N34.2 Other urethritis. Urethral meatitis. Ulcer of the urethra (external opening)
Urethritis:
NOS
postmenopausal
N34.3 Urethral syndrome, unspecified

N35 Urethral stricture

Excludes: urethral stricture after medical procedures ( N99.1)

N35.0 Post-traumatic urethral stricture
Urethral stricture:
postpartum
traumatic
N35.1 Post-infectious urethral stricture, not elsewhere classified
N35.8 Other urethral stricture
N35.9 Unspecified urethral stricture. External opening BDU

N36 Other diseases of the urethra

N36.0 Urethral fistula. False urethral fistula
Fistula:
urethroperineal
urethrorectal
urinary NOS
Excluded: fistula:
urethroscrotal ( N50.8)
urethrovaginal ( N82.1)
N36.1 Urethral diverticulum
N36.2 Urethral caruncle
N36.3 Prolapse of the urethral mucosa. Urethral prolapse. Urertocele in men
Excluded: urethrocele in women ( N81.0)
N36.8 Other specified diseases of the urethra
N36.9 Urethral disease, unspecified

N37* Lesions of the urethra in diseases classified elsewhere

N37.0* Urethritis in diseases classified elsewhere. Candidal urethritis ( B37.4+)
N37.8* Other lesions of the urethra in diseases classified elsewhere

N39 Other diseases of the urinary system

Excluded: hematuria:
NOS ( R31)
recurrent and persistent ( N02. -)
N02. -)
proteinuria NOS ( R80)

N39.0 Urinary tract infection without established localization
If it is necessary to identify the infectious agent, use an additional code ( B95-B97).
N39.1 Persistent proteinuria, unspecified
Excluded: complicating pregnancy, childbirth and the postpartum period ( O11-O15)
with refined morphological changes ( N06. -)
N39.2 Orthostatic proteinuria, unspecified
Excluded: with specified morphological changes ( N06. -)
N39.3 Involuntary urination
N39.4 Other specified types of urinary incontinence
When overflowing)
Reflex) urinary incontinence
upon awakening)
Excludes: enuresis NOS ( R32)
urinary incontinence:
NOS ( R32)
inorganic origin ( F98.0)
N39.8 Other specified diseases of the urinary system
N39.9 Urinary system disorder, unspecified

DISEASES OF THE MALE GENITAL ORGANS (N40-N51)

N40 Prostatic hyperplasia

Adenofibromatous hypertrophy)
Adenoma (benign)
Enlarged (benign) prostate
Fibroadenoma) glands
Fibroma)
Hypertrophy (benign)
Myoma
Adenoma of the median lobe (prostate)
Blockage of the prostate duct NOS
Excluded: benign tumors, except adenoma, fibroma
and prostate fibroids ( D29.1)

N41 Inflammatory diseases of the prostate gland

If it is necessary to identify the infectious agent, use an additional code ( B95-B97).

N41.0 Acute prostatitis
N41.1 Chronic prostatitis
N41.2 Prostate abscess
N41.3 Prostatocystitis
N41.8 Other inflammatory diseases of the prostate gland
N41.9 Inflammatory disease of the prostate, unspecified. Prostatitis NOS

N42 Other prostate diseases

N42.0 Prostate stones. Prostatic stone
N42.1 Congestion and hemorrhage in the prostate gland
N42.2 Prostate atrophy
N42.8 Other specified prostate diseases
N42.9 Prostate disease, unspecified

N43 Hydrocele and spermatocele

Includes: hydrocele of the spermatic cord, testis or tunica vaginalis
Excluded: congenital hydrocele ( P83.5)

N43.0 Hydrocele encyscum
N43.1 Infected hydrocele
If it is necessary to identify the infectious agent, use an additional code ( B95-B97).
N43.2 Other forms of hydrocele
N43.3 Hydrocele, unspecified
N43.4 Spermatocele

N44 Testicular torsion

Twist:
epididymis
spermatic cord
testicles

N45 Orchitis and epididymitis

If it is necessary to identify the infectious agent, use an additional code ( B95-B97).

N45.0 Orchitis, epididymitis and epididymo-orchitis with abscess. Abscess of the epididymis or testicle
N45.9 Orchitis, epididymitis and epididymo-orchitis without mention of abscess. Epididymitis NOS. Orchitis NOS

N46 Male infertility

Azoospermia NOS. Oligospermia NOS

N47 Excessive foreskin, phimosis and paraphimosis

Tightly fitting foreskin. Tight foreskin

N48 Other diseases of the penis

N48.0 Leukoplakia of the penis. Kraurosis of the penis
Excludes: carcinoma in situ of the penis ( D07.4)
N48.1 Balanoposthitis. Balanitis
If it is necessary to identify the infectious agent, use an additional code ( B95-B97).
N48.2 Other inflammatory diseases of the penis
Abscess)
Furuncle)
Carbuncle) of the corpus cavernosum and penis
Cellulite)
Cavernitis of the penis
If it is necessary to identify the infectious agent, use an additional code ( B95-B97).
N48.3 Priapism. Painful erection
N48.4 Impotence of organic origin
If necessary, an additional code is used to identify the cause.
Excludes: psychogenic impotence ( F52.2)
N48.5 Penile ulcer
N48.6 Balanitis. Plastic induration of the penis
N48.8 Other specific diseases of the penis
Atrophy)
Hypertrophy) of the corpus cavernosum and penis
Thrombosis)
N48.9 Disease of the penis, unspecified

N49 Inflammatory diseases of the male genital organs, not elsewhere classified

If it is necessary to identify the infectious agent, use an additional code ( B95-B97).
Excluded: inflammation of the penis ( N48.1-N48.2)
orchitis and epididymitis ( N45. -)

N49.0 Inflammatory diseases of the seminal vesicle. Vesiculitis NOS
N49.1 Inflammatory diseases of the spermatic cord, vaginal membrane and vas deferens. Vasit
N49.2 Inflammatory diseases of the scrotum
N49.8 Inflammatory diseases of other specified male genital organs
N49.9 Inflammatory diseases of the unspecified male genital organ
Abscess)
Furuncle) unspecified male
Carbuncle) of the genital organ
Cellulite)

N50 Other diseases of the male genital organs

Excluded: testicular torsion ( N44)

N50.0 Testicular atrophy
N50.1 Vascular disorders of the male genital organs
Hematocele)
Hemorrhage) of the male genital organs
Thrombosis)
N50.8 Other specific diseases of the male genital organs
Atrophy)
Hypertrophy) of the seminal vesicle, spermatic cord,
Swelling of the testicle [except atrophy], vaginal ulcer and vas deferens
Hylocele of the tunica vaginalis (non-filarial) NOS
Urethroscrotal fistula
Structure:
spermatic cord
vaginal membrane
vas deferens
N50.9 Male genital disease, unspecified

N51* Lesions of the male genital organs in diseases classified elsewhere

N51.0* Lesions of the prostate gland in diseases classified elsewhere
Prostatitis:
gonococcal ( A54.2+)
caused by Trichomonas ( A59.0+)
tuberculous ( A18.1+)
N51.1* Lesions of the testicle and its appendages in diseases classified in other headings
Chlamydial:
epididymitis ( A56.1+)
orchitis ( A56.1+)
Gonococcal:
epididymitis ( A54.2+)
orzit ( A54.2+)
Mumps orchitis ( B26.0+)
Tuberculosis:

• epididymis ( A18.1+)

• testicles ( A18.1+)

N51.2* Balanitis in diseases classified elsewhere
Balanitis:
amoebic ( A06.8+)
candida ( B37.4+)
N51.8* Other lesions of the male genital organs in diseases classified elsewhere
Filarial chylocele of the tunica vaginalis ( B74. -+)
Herpes infection of the male genital organs ( A60.0+)
Tuberculosis of the seminal vesicles ( A18.1+)

BREAST DISEASES (N60-N64)

Excluded: breast diseases associated with childbirth ( O91-O92)

N60 Benign breast dysplasia
Included: fibrocystic mastopathy
N60.0 Solitary cyst of the mammary gland. Breast cyst
N60.1 Diffuse cystic mastopathy. Cystic breast
Excluded: with epithelial proliferation ( N60.3)
N60.2 Fibroadenosis of the mammary gland
Excludes: breast fibroadenoma ( D24)
N60.3 Fibrosclerosis of the mammary gland. Cystic mastopathy with epithelial proliferation
N60.4 Breast duct ectasia
N60.8 Other benign breast dysplasias
N60.9 Benign breast dysplasia, unspecified

N61 Inflammatory diseases of the breast

Abscess (acute) (chronic) (not postpartum):
areola
mammary gland
Breast carbuncle
Mastitis (acute) (subacute) (not postpartum):
NOS
infectious
Excluded: infectious mastitis of the newborn ( P39.0)

N62 Breast hypertrophy

Gynecomastia
Breast hypertrophy:
NOS
massive pubescent

N63 Mass in the mammary gland, unspecified

Nodule(s) in the mammary gland NOS

N64 Other diseases of the breast

N64.0 Crack and fistula of the nipple
N64.1 Fat necrosis of the mammary gland. Fat necrosis (segmental) of the breast
N64.2 Breast atrophy
N64.3 Galactorrhea not associated with childbirth
N64.4 Mammalgia
N64.5 Other signs and symptoms of the breast. Induration of the mammary gland. Nipple discharge
Inverted nipple
N64.8 Other specified diseases of the breast. Galactocele. Subinvolution of the mammary gland (post-lactation)
N64.9 Breast disease, unspecified

INFLAMMATORY DISEASES OF FEMALE PELVIC ORGANS (N70-N77)

Excluded: complicating:
abortion, ectopic or molar pregnancy ( O00 -O07 , O08.0 )
pregnancy, childbirth and the postpartum period ( O23. — ,O75.3 , O85 , O86 . -)

N70 Salpingitis and oophoritis

Included: abscess:
fallopian tube
ovary
tubo-ovarian
pyosalpinx
salpingo-oophoritis
tubo-ovarian inflammatory disease
If it is necessary to identify the infectious agent, use an additional code ( B95-B97).

N70.0 Acute salpingitis and oophoritis
N70.1 Chronic salpingitis and oophoritis. Hydrosalpinx
N70.9 Salpingitis and oophoritis, unspecified

N71 Inflammatory diseases of the uterus, except the cervix

Includes: endo(myo)metritis
metritis
myometritis
pyometra
uterine abscess
If it is necessary to identify the infectious agent, use an additional code ( B95-B97).

N71.0 Acute inflammatory disease of the uterus
N71.1 Chronic inflammatory disease of the uterus
N71.9 Inflammatory disease of the uterus, unspecified

N72 Inflammatory disease of the cervix

Cervicitis)
Endocervicitis) with or without the presence of erosion or ectropion
Exocervicitis)
If necessary, identify the infectious agent
use additional code ( B95-B97).
Excluded: erosion and ectropion of the cervix without cervicitis ( N86)

N73 Other inflammatory diseases of the female pelvic organs

If it is necessary to identify the infectious agent, use an additional code ( B95-B97).

N73.0 Acute parametritis and pelvic cellulitis
Abscess:
broad ligament) specified as
parametrium) acute
Pelvic phlegmon in women)
N73.1 Chronic parametritis and pelvic cellulitis
N73.0, specified as chronic
N73.2 Parametritis and pelvic cellulitis, unspecified
Any condition in the subcategory N73.0, not specified as acute or chronic
N73.3 Acute pelvic peritonitis in women
N73.4 Chronic pelvic peritonitis in women
N73.5 Pelvic peritonitis in women, unspecified
N73.6 Pelvic peritoneal adhesions in women
Excluded: pelvic peritoneal adhesions in women postoperative ( N99.4)
N73.8 Other specified inflammatory diseases of the female pelvic organs
N73.9 Inflammatory diseases of the female pelvic organs, unspecified
Infectious or inflammatory diseases of the female pelvic organs NOS

N74* Inflammatory diseases of the female pelvic organs in diseases classified elsewhere

N74.0* Tuberculosis infection of the cervix ( A18.1+)
N74.1* Inflammatory diseases of the female pelvic organs of tuberculous etiology ( A18.1+)
Tuberculous endometritis
N74.2* Inflammatory diseases of the female pelvic organs caused by syphilis ( A51.4+, A52.7+)
N74.3* Gonococcal inflammatory diseases of the female pelvic organs ( A54.2+)
N74.4* Inflammatory diseases of the female pelvic organs caused by chlamydia ( A56.1+)
N74.8* Inflammatory diseases of the female pelvic organs in other diseases classified in other headings

N75 Diseases of the Bartholin gland

N75.0 Bartholin gland cyst
N75.1 Bartholin gland abscess
N75.8 Other diseases of the Bartholin gland. Bartholinitis
N75.9 Bartholin's gland disease, unspecified

N76 Other inflammatory diseases of the vagina and vulva

If it is necessary to identify the infectious agent, use an additional code ( B95-B97).
Excluded: senile (atrophic) vaginitis ( N95.2)

N76.0 Acute vaginitis. Vaginitis NOS
Vulvovaginitis:
NOS
spicy
N76.1 Subacute and chronic vaginitis

Vulvovaginitis:
chronic
subacute
N76.2 Acute vulvitis. Vulvitis NOS
N76.3 Subacute and chronic vulvitis
N76.4 Vulvar abscess. Furuncle of the vulva
N76.5 Vaginal ulceration
N76.6 Ulceration of the vulva
T76.8 Other specified inflammatory diseases of the vagina and vulva

N77* Ulceration and inflammation of the vulva and vagina in diseases classified elsewhere

NON-INFLAMMATORY DISEASES OF THE FEMALE GENITAL ORGANS (N80-N98)

N80 Endometriosis

N80.0 Endometriosis of the uterus. Adenomyosis
N80.1 Ovarian endometriosis
N80.2 Fallopian tube endometriosis
N80.3 Endometriosis of the pelvic peritoneum
N80.4 Endometriosis of the rectovaginal septum and vagina
N80.5 Intestinal endometriosis
N80.6 Endometriosis of the skin scar
N80.8 Other endometriosis
N80.9 Endometriosis, unspecified

N81 Prolapse of female genital organs

Excluded: genital prolapse complicating pregnancy, labor or delivery ( O34.5)
prolapse and hernia of the ovary and fallopian tube ( N83.4)
prolapse of the vaginal stump (vault) after hysterectomy ( N99.3)

N81.0 Urethrocele in women

Excluded: urethrocele with:
cystocele ( N81.1)
uterine prolapse ( N81.2-N81.4)
N81.1 Cystocele. Cystocele with urethrocele. Prolapse of the (anterior) vaginal wall NOS
Excluded: cystotele with uterine prolapse ( N81.2-N81.4)
N81.2 Incomplete prolapse of the uterus and vagina. Cervical prolapse NOS
Vaginal prolapse:
first degree
second degree
N81.3 Complete prolapse of the uterus and vagina. Prosidence (uterus) NOS. Third degree uterine prolapse
N81.4 Unspecified uterine and vaginal prolapse. Uterine prolapse NOS
N81.5 Enterocele of the vagina
Excluded: enterocele with uterine prolapse ( N81.2-N81.4)
N81.6 Rectocele. Prolapse of the posterior vaginal wall
Excluded: rectal prolapse ( K62.3)
rectocele with uterine prolapse ( N81.2-N81.4)
N81.8 Other forms of female genital organ prolapse. Insufficiency of the pelvic floor muscles
Old pelvic floor muscle tears
N81.9 Female genital prolapse, unspecified

N82 Fistulas involving the female genital organs

Excluded: vesicointestinal fistula ( N32.1)

N82.0 Vesicovaginal fistula
N82.1 Other fistulas of the female genitourinary tract
Fistulas:
cervicovesical
ureterovaginal
urethrovaginal
utero-ureteric
uterovesical
N82.2 Vaginal-small intestinal fistula
N82.3 Vaginal-colic fistula. Rectovaginal fistula
N82.4 Other enterogenital fistulas in women. Intestinal-uterine fistula
N82.5 Genital-cutaneous fistulas in women

Fistula:
uteroabdominal
vagina-perineal
N82.8 Other female genital fistulas
N82.9 Female genital fistula, unspecified

N83 Non-inflammatory lesions of the ovary, fallopian tube and broad ligament of the uterus

Excluded: hydrosalpinx ( N70.1)

N83.0 Follicular ovarian cyst. Graafian follicle cyst. Hemorrhagic follicular cyst (ovarian)
N83.1 Corpus luteum cyst. Hemorrhagic cyst of the corpus luteum
N83.2 Other and unspecified ovarian cysts
Retention cyst)
Simple cyst) of the ovary
Excluded: ovarian cyst:
associated with developmental anomaly ( Q50.1)
neoplastic ( D27)
polycystic ovary syndrome ( E28.2)
N83.3 Acquired atrophy of the ovary and fallopian tube
N83.4 Prolapse and hernia of the ovary and fallopian tube
N83.5 Torsion of the ovary, ovarian stalk and fallopian tube
Twist:
additional pipe
Morgagni cysts
N83.6 Hematosalpinx
Excluded: hematosalpinx with:
hematocolposome ( N89.7)
hematometer ( N85.7)
N83.7 Hematoma of the broad ligament of the uterus
N83.8 Other non-inflammatory diseases of the ovary, fallopian tube and broad ligament of the uterus
[Masters-Allen] broad ligament rupture syndrome
N83.9 Non-inflammatory disease of the ovary, fallopian tube and broad ligament of the uterus, unspecified

N84 Polyp of female genital organs

Excluded: adenomatous polyp ( D28. -)
placental polyp ( O90.8)

N84.0 Polyp of the uterine body
Polyp:
endometrium
uterus NOS
Excludes: polypoid endometrial hyperplasia ( N85.0)
N84.1 Cervical polyp. Polyp of the cervical mucosa
N84.2 Vaginal polyp
N84.3 Vulvar polyp. Labia polyp
N84.8 Polyp of other parts of the female genital organs
N84.9 Female genital polyp, unspecified

N85 Other non-inflammatory diseases of the uterus, excluding the cervix

Excludes: endometriosis ( N80. -)
inflammatory diseases of the uterus ( N71. -)

non-inflammatory diseases of the cervix ( N86-N88)
uterine body polyp ( N84.0)
uterine prolapse ( N81. -)

N85.0 Glandular hyperplasia of the endometrium
Endometrial hyperplasia:
NOS
cystic
glandular-cystic
polypoid
N85.1 Adenomatous endometrial hyperplasia. Atypical endometrial hyperplasia (adenomatous)
N85.2 Uterine hypertrophy. Large or enlarged uterus
Excludes: postpartum uterine hypertrophy ( O90.8)
N85.3 Subinvolution of the uterus
Excludes: postpartum subinvolution of the uterus ( O90.8)
N85.4 Incorrect position of the uterus
Anteversion)
Retroflexion) of the uterus
Retroversion)
Excluded: as a complication of pregnancy, childbirth or postpartum period ( O34.5, O65.5)
N85.5 Inversion of the uterus
O71.2)
postpartum uterine prolapse ( N71.2)
N85.6 Intrauterine synechiae
N85.7 Hematometra. Hematosalpinx with hematometra
Excludes: hematometra with hematocolpos ( N89.7)
N85.8 Other specified inflammatory diseases of the uterus. Acquired uterine atrophy. Uterine fibrosis NOS
N85.9 Non-inflammatory disease of the uterus, unspecified. Uterine lesions NOS

N86 Erosion and ectropion of the cervix

Decubital (trophic) ulcer)
Inversion) of the cervix
Excluded: with cervicitis ( N72)

N87 Cervical dysplasia

Excludes: carcinoma in situ of the cervix ( D06. -)

N87.0 Mild cervical dysplasia. Cervical intraepithelial neoplasia grade I
N87.1 Moderate cervical dysplasia. Cervical intraepithelial neoplasia grade II
N87.2 Severe cervical dysplasia, not classified elsewhere
Severe dysplasia NOS
Excludes: cervical intraepithelial neoplasia grade III with or without mention
D06. -)
N87.9 Cervical dysplasia, unspecified

N88 Other non-inflammatory diseases of the cervix

Excluded: inflammatory diseases of the cervix ( N72)
cervical polyp ( N84.1)

N88.0 Leukoplakia of the cervix
N88.1 Old cervical ruptures. Cervical adhesions
O71.3)
N88.2 Cervical stricture and stenosis
Excluded: as a complication of childbirth ( O65.5)
N88.3 Cervical insufficiency
Examination and assistance for (suspected) isthmic-cervical insufficiency outside pregnancy
Excluded: complicating the condition of the fetus and newborn ( P01.0)
complicating pregnancy ( O34.3)
N88.4 Hypertrophic lengthening of the cervix
N88.8 Other specified non-inflammatory diseases of the cervix
Excluded: current obstetric trauma ( O71.3)
N88.9 Non-inflammatory disease of the cervix, unspecified

Excluded: carcinoma in situ of the vagina ( D07.2), inflammation of the vagina ( N76. -), senile (atrophic) vaginitis ( N95.2)
leucorrhoea with trichomoniasis ( A59.0)
N89.0 Mild vaginal dysplasia. Vaginal intraepithelial neoplasia grade I
N89.1 Moderate vaginal dysplasia. Vaginal intraepithelial neoplasia grade II
N89.2 Severe vaginal dysplasia, not classified elsewhere
Severe vaginal dysplasia NOS
Excludes: vaginal intraepithelial neoplasia grade III with or without mention
about pronounced dysplasia ( D07.2)
N89.3 Vaginal dysplasia, unspecified
N89.4 Vaginal leukoplakia
N89.5 Vaginal stricture and atresia
Vaginal:
adhesions
stenosis
Excluded: postoperative vaginal adhesions ( N99.2)
N89.6 Dense hymen. Rigid hymen. Tight virgin ring
Excluded: hymen closed ( Q52.3)
N89.7 Hematocolpos. Hematocolpos with hematometra or with hematosalpinx
N89.8 Other non-inflammatory diseases of the vagina. Beli NOS. Old vaginal rupture. Vaginal ulcer
Excluded: current obstetric trauma ( O70. — , O71.4,O71.7-O71.8)
old tear involving the pelvic floor muscles ( N81.8)
N89.9 Non-inflammatory disease of the vagina, unspecified

N90 Other non-inflammatory diseases of the vulva and perineum

Excluded: carcinoma in situ of the vulva ( D07.1)
current obstetric trauma ( O70. — , O71.7-O71.8)
inflammation of the vulva ( N76. -)

N90.0 Mild vulvar dysplasia. Vulvar intraepithelial neoplasia grade I
N90.1 Moderate vulvar dysplasia. Vulvar intraepithelial neoplasia grade II
N90.2 Severe vulvar dysplasia, not elsewhere classified
Severe vulvar dysplasia NOS
Excludes: vulvar intraepithelial neoplasia grade III with or without mention
about pronounced dysplasia ( D07.1)
N90.3 Vulvar dysplasia, unspecified
N90.4 Leukoplakia of the vulva
Dystrophy)
Kraurosis) of the vulva
N90.5 Vulvar atrophy. Vulvar stenosis
N90.6 Vulvar hypertrophy. Hypertrophy of the labia
N90.7 Vulvar cyst
N90.8 Other specified non-inflammatory diseases of the vulva and perineum. Vulvar adhesions. Clitoral hypertrophy
N90.9 Non-inflammatory disease of the vulva and perineum, unspecified

N91 Absence of menstruation, scanty and infrequent menstruation

Excludes: ovarian dysfunction ( E28. -)

N91.0 Primary amenorrhea. Irregular menstruation during puberty
N91.1 Secondary amenorrhea. Lack of menstruation in women who previously had them
N91.2 Amenorrhea, unspecified. Absence of menstruation NOS
N91.3 Primary oligomenorrhea. Scanty or infrequent menstruation from the beginning of their appearance
N91.4 Secondary oligomenorrhea. Scanty or infrequent periods in women with previously normal periods
N91.5 Oligomenorrhea, unspecified. Hypomenorrhea NOS

N92 Heavy, frequent and irregular menstruation

Excluded: bleeding after menopause ( N95.0)

N92.0 Heavy and frequent menstruation with a regular cycle
Periodically heavy menstruation NOS. Menorrhagia NOS. Polymenorrhea
N92.1 Heavy and frequent menstruation with an irregular cycle
Irregular bleeding between menstrual periods
Irregular, shortened intervals between menstrual bleeding. Menometrorrhagia. Metrorrhagia
N92.2 Heavy menstruation during puberty
Heavy bleeding at the beginning of the menstrual period. Pubertal menorrhagia. Pubertal bleeding
N92.3 Ovulatory bleeding. Regular menstrual bleeding
N92.4 Heavy bleeding in the premenopausal period
Menorrhagia or metrorrhagia:
menopausal
in menopause
premenopausal
premenopausal
N92.5 Other specified forms of irregular menstruation
N92.6 Irregular menstruation, unspecified
Irregular:
bleeding NOS
menstrual cycles NOS
Excluded: irregular menstruation due to:
prolonged intervals or scanty bleeding ( N91.3-N91.5)
shortened intervals or excessive bleeding ( N92.1)

N93 Other abnormal bleeding from the uterus and vagina

Excluded: neonatal vaginal bleeding ( P54.6)
false menstruation ( P54.6)

N93.0 Postcoital or contact bleeding
N93.8 Other specified abnormal bleeding from the uterus and vagina
Dysfunctional or functional uterine or vaginal bleeding NOS
N93.9 Abnormal uterine and vaginal bleeding, unspecified

N94 Pain and other conditions associated with the female genital organs and the menstrual cycle

N94.0 Pain in the middle of the menstrual cycle
N94.1 Dyspareunia
Excludes: psychogenic dyspareunia ( F52.6)
N94.2 Vaginismus
Excluded: psychogenic vaginismus ( F52.5)
N94.3 Premenstrual tension syndrome
N94.4 Primary dysmenorrhea
N94.5 Secondary dysmenorrhea
N94.6 Dysmenorrhea, unspecified
N94.8 Other specified conditions related to the female genital organs and menstrual cycle
N94.9 Conditions related to the female genital organs and menstrual cycle, unspecified

N95 Menopausal and other perimenopausal disorders

Excluded: heavy bleeding in the premenopausal period ( N92.4)
postmenopausal:
osteoporosis ( M81.0)
with a pathological fracture ( M80.0)
urethritis ( N34.2)
premature menopause NOS ( E28.3)

N95.0 Postmenopausal bleeding
N95.3)
N95.1 Menopause and menopause in women
Menopause-related symptoms such as hot flashes, insomnia, headaches, attention problems
Excluded: associated with artificial menopause ( N95.3)
N95.2 Postmenopausal atrophic vaginitis. Senile (atrophic) vaginitis
Excluded: associated with artificial menopause ( N95.3)
N95.3 Conditions associated with artificially induced menopause. Syndrome after artificial menopause
N95.8 Other specified disorders of menopause and perimenopause
N95.9 Menopausal and perimenopausal disorders, unspecified

N96 Recurrent miscarriage

Examination or provision of medical care outside of pregnancy. Relative infertility
Excluded: current pregnancy ( O26.2)
with current abortion ( O03-O06)

N97 Female infertility

Includes: inability to become pregnant
female sterility NOS
Excluded: relative infertility ( N96)

N97.0 Female infertility associated with lack of ovulation
N97.1 Female infertility of tubal origin. Associated with congenital fallopian tube anomaly
Pipe:
obstruction
blockage
stenosis
N97.2 Female infertility of uterine origin. Associated with congenital uterine anomaly
Egg implantation defect
N97.3 Female infertility of cervical origin
N97.4 Female infertility associated with male factors
N97.8 Other forms of female infertility
N97.9 Female infertility, unspecified

N98 Complications associated with artificial insemination

N98.0 Infection associated with in vitro fertilization
N98.1 Ovarian hyperstimulation
Ovarian hyperstimulation:
NOS
associated with induced ovulation
N98.2 Complications associated with an attempt to implant a fertilized egg after extracorporeal
fertilization
N98.3 Complications associated with attempted embryo implantation
N98.8 Other complications associated with artificial insemination
Complications of artificial insemination:
donor sperm
husband's sperm
N98.9 Complications associated with artificial insemination, unspecified

OTHER DISEASES OF THE GINOROGENITAL SYSTEM (N99)

N99 Disorders of the genitourinary system after medical procedures, not elsewhere classified

Excluded: radiation cystitis ( N30.4)
osteoporosis after surgical removal of the ovary ( M81.1)
with a pathological fracture ( M80.1)
conditions associated with artificially induced menopause ( N95.3)

N99.0 Postoperative renal failure
N99.1 Postoperative urethral stricture. Urethral stricture after catheterization
N99.2 Postoperative vaginal adhesions
N99.3 Prolapse of the vaginal vault after hysterectomy
N99.4 Postoperative adhesions in the pelvis
N99.5 Dysfunction of the external stoma of the urinary tract
N99.8 Other disorders of the genitourinary system after medical procedures. Residual ovarian syndrome
N99.9 Disorders of the genitourinary system after medical procedures, unspecified

Glomerular diseases are a group of pathologies that have similar functional, structural and clinical features and occur with primary damage to the glomeruli of the kidneys. Their classification is based on division according to the leading syndrome - nephritic, nephrotic or hematuric. How is glomerulonephritis coded according to ICD 10?

Basics of medical classification

The main purpose of the International Classification of Diseases is the systematic recording, analysis, interpretation and comparison of data on diseases and health-related problems among residents of different countries. Short alphanumeric codes replace long and difficult to pronounce names of various pathologies in documentation for doctors from all over the world. This allows for concise and effective reports on the incidence, prevalence and mortality of any health problem known to mankind.

According to ICD 10, all diseases are conditionally divided into 21 classes according to the predominant damage to organs. Thus, pathologies of the kidneys and urinary tract belong to class XIV.

Glomerulonephritis: clinical and morphological features

Glomerulonephritis is not a separate disease, but a whole group of pathologies united by a number of common features that reflect the kidney’s response to an infectious and inflammatory lesion. The main pathogenetic moment in the development of GN is damage to the renal glomeruli. It leads to the following violations:

• isolated urinary syndrome – proteinuria, hematuria of varying severity;
• oliguria – decrease in the volume of daily diuresis;
• decreased glomerular filtration rate.

As the disease progresses, inflammation of the interstitium and functional disorders of the tubules occur. In the clinical picture, this is manifested by disturbances in ion transport and a decrease in the concentrating ability of the kidneys. The terminal stage of the disease is accompanied by renal failure and uremia.

How is the disease classified according to the ICD?

All glomerular diseases in the ICD have alphanumeric codes starting with the Latin letter N:

• N00 – acute nephritic syndrome (including acute glomerulonephritis);
• N01 – rapidly progressive nephritic syndrome (nephritis, glomerulonephritis and other forms of glomerular disease with a corresponding course);
• N02 – persistent recurrent hematuria;
• N03 – chronic nephritic syndrome (including CGN);
• N04 – nephrotic syndrome (including lipoid nephrosis, a congenital form of pathology);
• N05 – nephritic syndrome (glomerulonephritis), unspecified;
• N06 – proteinuria (isolated);
• N07 – hereditary forms of nephropathy (Alport disease, amyloid nephropathy, familial amyloidosis).

The most common forms of pathology include acute and chronic glomerulonephritis.

Acute glomerulonephritis is coded as N00. This pathology is based on an infectious-allergic process: an attack of the glomeruli of the kidney by immune complexes “antigen-antibody” formed as a result of bacterial (usually streptococcal) or viral infection.

Chronic glomerulonephritis has ICD 10 code N03. It is characterized by progressive diffuse damage to the functional apparatus of the kidneys, resulting in their sclerosis and failure. Formed as a consequence of acute symptoms with absent or ineffective treatment.

If necessary, the above code is supplemented with a third order of numbers indicating the clinical and morphological features of the disease. Acute or chronic glomerulonephritis occurs with:

• minor changes(.0);
• focal (segmental) changes – hyalinosis, sclerosis (.1);
• diffuse membranous changes (.2);
• diffuse mesangial proliferative changes (.3);
• diffuse endocapillary proliferative changes (.4);
• diffuse mesangiocapillary changes (.5).
• extracapillary changes (.7).

Additionally, there is a special form of diffuse mesangiocapillary glomerulonephritis - dense sediment disease (.6). Glomerular inflammation with other changes is coded .8, unspecified - .9.